Author

E. Schadt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:genome assembly· 2 Assay:scRNA-seq· 2 Research Focus:variant calling· 2 Assay:whole-genome sequencing· 2

Frequent Co-Authors

R. Sebra· 2 shared A. Bashir· 1 shared A. Greenstein· 1 shared A. Kasarskis· 1 shared A. Leader· 1 shared A. Rahman· 1 shared A. Rendo· 1 shared A. Yadaw· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

R. Sebra

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A Library Of Induced Pluripotent Stem Cells From Clinically Well-Characterized, Diverse Healthy Human Individuals

C. Schaniel, P. Dhanan, B. Hu, Y. Xiong, T. Raghunandan, D. M. Gonzalez, S. L. D'Souza, A. Yadaw, J. Hansen, G. Jayaraman, B. Mathew, M. Machado, S. Berger, J. Tripodi, V. Najfeld, , R. Iyengar

The paper generates and characterizes a diverse library of hiPSCs from healthy humans for use in disease modeling and drug response studies.

bioRxiv · Oct 29, 2020Cell Biology

Complete genome screening of clinical MRSA isolates identifies lineage diversity and provides full resolution of transmission and outbreak events

M. J. Sullivan, D. R. Altman, K. I. Chacko, B. Ciferri, E. Webster, T. R. Pak, G. Deikus, M. Lewis-Sandari, Z. Khan, C. Beckford, A. Rendo, F. Samaroo, R. Sebra, R. Karam-Howlin, T. Dingle, , H. van Bakel

This study uses long-read sequencing to analyze MRSA genomes, revealing lineage diversity and detailed transmission dynamics in an outbreak.

bioRxiv · May 21, 2019 · PreprintMicrobiology

Single-cell analysis of Crohn’s disease lesions identifies a pathogenic cellular module associated with resistance to anti-TNF therapy

J. C. Martin, G. Boschetti, C. Chang, R. Ungaro, M. Giri, L.-S. Chuang, S. Nayar, A. Greenstein, M. Dubinsky, L. Walker, A. Leader, J. Fine, C. Whitehurst, L. Mbow, S. Kugathasan, , E. Kenigsberg

bioRxiv · Dec 20, 2018Immunology

B cell defects observed in Nod2 knockout mice are a consequence of a Dock2 mutation frequently found in inbred strains

S.-Y. Wong, M. Coffre, D. Ramanan, L. E. Gomez, L. Peters, E. Schadt, S. Koralov, K. Cadwell

The paper identifies a Dock2 mutation in Nod2-/- mice that causes B cell defects, independent of Nod2 deletion, and shows the mutation is common in inbred strains.

bioRxiv · Jan 4, 2018Immunology