Author

S. Koralov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:genome assembly· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:variant calling· 1

Frequent Co-Authors

A. Anderson· 1 shared A. Herrera· 1 shared A. Mohamed· 1 shared A. Schinlever· 1 shared A. Valero-Jimenez· 1 shared C. Steppan· 1 shared D. Ramanan· 1 shared E. Ivanova· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperMicrobiology

A. Anderson

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A comparative analysis of SARS-CoV-2 antivirals in human airway models characterizes 3CLpro inhibitor PF-00835231 as a potential new treatment for COVID-19

M. de Vries, A. Mohamed, R. A. Prescott, A. Valero-Jimenez, L. Desvignes, R. O'Connor, C. Steppan, J. C. Devlin, E. Ivanova, A. Herrera, A. Schinlever, P. Loose, K. Ruggles, S. Koralov, A. Anderson, , M. Dittmann

This study compares the antiviral efficacy of PF-00835231, GC-376, and remdesivir in human airway models, showing PF-00835231 is at least as potent as the others and not affected by P-glycoprotein efflux.

bioRxiv · Aug 28, 2020 · PreprintMicrobiology

B cell defects observed in Nod2 knockout mice are a consequence of a Dock2 mutation frequently found in inbred strains

S.-Y. Wong, M. Coffre, D. Ramanan, L. E. Gomez, L. Peters, E. Schadt, S. Koralov, K. Cadwell

The paper identifies a Dock2 mutation in Nod2-/- mice that causes B cell defects, independent of Nod2 deletion, and shows the mutation is common in inbred strains.

bioRxiv · Jan 4, 2018Immunology