Author

E. Yau

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

T. M. Umstead· 2 shared Z. C. Chroneos· 2 shared A. C. Arnold· 1 shared A. Erozenci· 1 shared A. Valles Marti· 1 shared C. R. Jimenez· 1 shared C. Song· 1 shared D. Dekker· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersPhysiology

T. M. Umstead

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Large pan-cancer cell screen coupled to (phospho-) proteomics underscores high-dose vitamin C as a potent anti-cancer agent

A. Valles Marti, F. Böttger, E. Yau, K. Tejjani, L. Meijs, S. Sharma, M. Mumtaz, T. Le Large, A. Erozenci, D. Dekker, T. Schelfhorst, J. P. Medema, I. V. Bijnsdorp, J. C. Knol, S. R. Piersma, , C. R. Jimenez

This study evaluates high-dose vitamin C's anti-cancer effects across 51 cancer cell lines, finding high sensitivity and proteomic changes linked to cell cycle and immune pathways.

bioRxiv · Dec 19, 2023 · PreprintCancer Biology

Disruption of the surfactant protein A receptor SP-R210L (CD245α/MYO18Aα) alters respiratory function and iron sequestration in alveolar macrophages of aged mice

E. Yau, T. M. Umstead, R. Abdulqadir, K. Fino, Z. Guan, S. Hu, S. Diangelo, K. Hassan, S. Bingaman, H. M. Atkins, T. K. Cooper, A. C. Arnold, E. S. Halstead, Z. C. Chroneos

This study shows that disrupting the SP-R210L isoform in aged mice impairs respiratory function and iron sequestration in alveolar macrophages, leading to chronic dysregulation and increased interstitial lung disease risk.

bioRxiv · Apr 14, 2021 · PreprintPhysiology

Genomic and epigenomic adaptation in SP-R210 (Myo18A) isoform-deficient macrophages

E. Yau, Y. Chen, C. Song, J. Web, M. Carillo, Y. I. Kawasawa, Z. Tang, Y. Takahashi, T. M. Umstead, S. Dovat, Z. C. Chroneos

This study investigates how the absence of the SP-R210L isoform in macrophages leads to a hyper-inflammatory state through altered genomic and epigenomic responses.

bioRxiv · Apr 2, 2021Immunology