Author

G. C. Li

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Ecology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:ecology & biodiversity· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. C. Koong· 1 shared A. Diep· 1 shared A. J. Giaccia· 1 shared A. K. Kenworthy· 1 shared C. Cenik· 1 shared C. Jia· 1 shared C. R. Sanders· 1 shared D. Jiang· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. C. Koong

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Woody species with high wood density are more vulnerable to global change in South China

G. C. Li, S. D. Veresoglou

The paper assesses distribution shifts of eight rare woody species in South China under climate change and identifies wood density as a trait predicting vulnerability to global change.

bioRxiv · Nov 6, 2023Ecology

Developing High-Affinity Decoy Receptor Optimized for Multiple Myeloma and Diffuse Large B Cell Lymphoma Treatment

Y. R. Miao, K. Thakkar, C. Cenik, D. Jiang, K. Mizuno, C. Jia, G. C. Li, H. Zhao, A. Diep, J. Y. Xu, X. E. Zhang, T. T. C. Yang, M. Liedtke, P. Abidi, W.-s. Leung, , A. J. Giaccia

This paper develops an engineered sBCMA-Fc fusion protein with enhanced binding affinity to APRIL and BAFF, showing improved antitumor activity against multiple myeloma and diffuse large B cell lymphoma in preclinical models.

bioRxiv · May 3, 2021Cancer Biology

Peripheral Myelin Protein 22 Preferentially Partitions into Ordered Phase Membrane Domains

J. T. Marinko, G. C. Li, A. K. Kenworthy, C. R. Sanders

This paper shows that PMP22 preferentially partitions into ordered membrane domains, and disease mutations disrupt this preference.

bioRxiv · Jan 28, 2020Biophysics