Author

G. Patti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Eisenbarth· 1 shared A. J. Lutkewitte· 1 shared A. LaPoint· 1 shared A. Schulze· 1 shared B. N. Finck· 1 shared C. L. Stallings· 1 shared D. Ferguson· 1 shared E. Buglakova· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. Eisenbarth

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Spatial single-cell isotope tracing reveals heterogeneity of de novo fatty acid synthesis in cancer cells

E. Buglakova, M. Ekelöf, M. Schwaiger-Haber, L. Schlicker, M. R. Molenaar, S. Mohammed, L. Stuart, A. Eisenbarth, V. Hilsenstein, G. Patti, A. Schulze, M. Snaebjornsson, T. Alexandrov

This paper introduces 13C-SpaceM, a method for spatial single-cell isotope tracing of de novo fatty acid synthesis in cancer cells, revealing metabolic heterogeneity in liver and glioma tissues.

bioRxiv · Aug 18, 2023 · PreprintBiochemistry

Mitochondrial Pyruvate Carrier Inhibition Attenuates Hepatic Stellate Cell Activation and Liver Injury in a Mouse Model of Metabolic Dysfunction-associated Steatotic Liver Disease

M. Habibi, D. Ferguson, S. J. Eichler, M. M. Chan, A. LaPoint, T. M. Shew, M. He, A. J. Lutkewitte, J. D. Schilling, K. Y. Cho, G. Patti, B. N. Finck

bioRxiv · Feb 13, 2023 · PreprintPhysiology

Inducing vulnerability to InhA inhibition restores isoniazid susceptibility in drug resistant Mycobacterium tuberculosis

G. A. Harrison, K. Cho, E. R. Wang, S. Sarkar, F. Almqvist, G. Patti, C. L. Stallings

This paper identifies a compound, C10, that re-sensitizes drug-resistant Mycobacterium tuberculosis to isoniazid by exploiting a metabolic vulnerability, without altering InhA activity or INH-NAD levels.

bioRxiv · Feb 6, 2023 · PreprintMicrobiology