Author

H. Christofk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:human· 2 Research Focus:metabolism & bioenergetics· 2 Research Focus:cell signaling & trafficking· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

A. Delcourt· 1 shared A. Flores· 1 shared A. Jones· 1 shared A. Lai· 1 shared A. Petcherski· 1 shared A. S. Divakaruni· 1 shared A. S. Divakurani· 1 shared A. S. Goldstein· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperCancer Biology

A. Delcourt

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3 papers

Recent·Most cited

Defining metabolic flexibility in hair follicle stem cell induced squamous cell carcinoma

C. Galvan, A. Flores, V. Cerillos, I. Avila, C. Murphy, W. Zheng, T. T. To, H. Christofk, W. Lowry

This study investigates metabolic flexibility in hair follicle stem cell-derived squamous cell carcinoma by blocking glycolysis and glutaminolysis, showing tumors can compensate by upregulating lactate consumption.

bioRxiv · Oct 16, 2023Cancer Biology

Androgen receptor inhibition induces metabolic reprogramming and increased reliance on oxidative mitochondrial metabolism in prostate cancer

P. Crowell, J. Giafaglione, A. Jones, N. Nunley, T. Hashimoto, A. Delcourt, A. Petcherski, M. Bernard, R. R. Huang, J.-Y. Low, N. Matulionis, X. Guan, N. Navone, J. Alumkal, M. Haffner, , A. S. Goldstein

This study shows that androgen receptor inhibition in prostate cancer cells shifts metabolism to oxidative mitochondrial pathways and increases sensitivity to complex I inhibitors.

Cell Reports · Oct 1, 2023Peer-reviewedCancer Biology

Glioblastoma utilizes fatty acids and ketone bodies for growth allowing progression during ketogenic diet therapy

J. Sperry, M. C. Condro, L. Guo, K. K. O. Kim, N. Vanderveer-Harris, D. Braas, W. B. Pope, A. S. Divakurani, A. Lai, H. Christofk, J. E. Le Belle, H. I. Kornblum

This study shows that glioblastoma uses fatty acids and ketone bodies for growth, and that ketogenic diet therapy does not effectively inhibit tumor progression.

bioRxiv · Aug 11, 2020Cancer Biology