Author

H. Katsuno-Kambe

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. S. Yap· 2 shared R. J. Ju· 2 shared B. Ladoux· 1 shared B. R. Acharya· 1 shared G. A. Gomez· 1 shared I. Noordstra· 1 shared J. E. Hudson· 1 shared J. L. C. Teo· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersCell Biology

A. S. Yap

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Spontaneous cell morphogenesis via self-propelled actin filaments

K. Yagami, K. Baba, T. Minegishi, S. Misu, H. Katsuno-Kambe, K. Okano, Y. Sakumura, Y. Hosokawa, N. Inagaki

The paper shows actin filaments move directionally via treadmilling, driving membrane protrusions and cell migration through physical mechanisms.

bioRxiv · Feb 28, 2024Cell Biology

Collagen polarization promotes epithelial elongation by stimulating locoregional cell proliferation.

H. Katsuno-Kambe, J. L. Teo, R. J. Ju, J. E. Hudson, S. J. Stehbens, A. S. Yap

The paper shows collagen polarization drives epithelial elongation by inducing localized cell proliferation through β1-integrin and ERK signaling.

bioRxiv · Feb 28, 2021Cell Biology

Caveolae set levels of epithelial monolayer tension to eliminate tumor cells

J. L. C. Teo, G. A. Gomez, I. Noordstra, S. Verma, V. Tomatis, B. R. Acharya, L. Balasubramaniam, H. Katsuno-Kambe, R. Templin, K.-A. McMahon, R. J. Ju, S. J. Stebhens, B. Ladoux, R. G. Parton, A. S. Yap

The paper shows caveolae regulate epithelial tension through lipid signaling, enabling the elimination of oncogene-transformed cells by apical extrusion.

bioRxiv · May 9, 2019 · PreprintCell Biology