Author

H. M. Kudyba

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Research Focus:genome assembly· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

D. W. Cobb· 3 shared V. Muralidharan· 3 shared A. Florentin· 2 shared M. Krakowiak· 2 shared A. Villegas· 1 shared B. Bruton· 1 shared B. Singh· 1 shared D. Ljolje· 1 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersCell Biology

D. W. Cobb

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Author profile →

3 papers

Recent·Most cited

A druggable oxidative folding pathway in the endoplasmic reticulum of human malaria parasites

D. W. Cobb, H. M. Kudyba, A. Villegas, M. R. Hoopmann, R. Baptista, B. Bruton, M. Krakowiak, R. L. Moritz, V. Muralidharan

This study identifies a druggable oxidative folding pathway in the ER of P. falciparum, showing that PfPDI8 is essential and sensitive to small molecule inhibition.

bioRxiv · May 13, 2020Microbiology

The ER chaperone PfGRP170 is essential for asexual development and is linked to stress response in malaria parasites.

H. M. Kudyba, D. W. Cobb, M. A. Fierro, A. Florentin, D. Ljolje, B. Singh, N. W. Lucchi, V. Muralidharan

This study identifies PfGRP170 as an essential ER chaperone in Plasmodium falciparum, critical for asexual development and stress response.

bioRxiv · Apr 17, 2019Cell Biology

CRISPR/Cas9 gene editing to make conditional mutants of the human malaria parasite Plasmodium falciparum

H. M. Kudyba, D. W. Cobb, A. Florentin, M. Krakowiak, V. Muralidharan

This paper describes a CRISPR/Cas9 method for generating conditional knockdown mutants in Plasmodium falciparum, enabling genetic manipulation of the malaria parasite.

bioRxiv · May 15, 2018Cell Biology