Author

H. M. Marriott

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. M. Condliffe· 1 shared C. D. Russell· 1 shared C. G. Wright· 1 shared C. J. Hill· 1 shared C. Pridans· 1 shared D. H. Dockrell· 1 shared E. C. Prestwich· 1 shared E. Fisk· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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1 shared paperImmunology

A. M. Condliffe

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3 papers

Recent·Most cited

The EGFR/ErbB inhibitor neratinib modifies the neutrophil phosphoproteome and promotes apoptosis and clearance by airway macrophages

K. D. Herman, C. G. Wright, H. M. Marriott, S. C. McCaughran, M. Collins, S. A. Renshaw, L. R. Prince

This paper shows neratinib induces neutrophil apoptosis and enhances macrophage clearance in acute lung injury, supported by phosphoproteomics analysis.

bioRxiv · Apr 14, 2022Immunology

Mitochondrial fission is increased in macrophages during mROS production in response to S. pneumoniae

M. Mohasin, K. Balbirnie-Cumming, E. Fisk, E. C. Prestwich, C. D. Russell, J. Marshall, C. Pridans, S. P. Allen, P. J. Shaw, K. J. De Vos, C. J. Hill, P. Bullough, A. M. Condliffe, T. J. Mitchell, H. M. Marriott, D. H. Dockrell

This study shows that mitochondrial fission in macrophages increases during mROS production in response to S. pneumoniae, linked to reduced oxidative phosphorylation and enhanced bacterial killing.

bioRxiv · Nov 16, 2021 · PreprintImmunology

The mouse lung early cellular innate immune response is not sufficient to control fungal infection with Cryptococcus neoformans

J. Rudman, H. M. Marriott, L. M. Carlin, S. A. Johnston

This study uses an ex vivo mouse lung model to show that innate immune cells cannot control Cryptococcus neoformans infection and provide an intracellular niche for fungal growth.

bioRxiv · Jun 24, 2019 · PreprintImmunology