Author

Heather L. Stickney

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:zebrafish· 3

Frequent Co-Authors

Florencia CavodeassiiD· 3 shared Quenten SchwarziD· 3 shared Rodrigo YoungiD· 3 shared Stephen W. WilsoniD· 3 shared Thomas A. HawkinsiD· 3 shared Gaia GestriiD· 2 shared Allison Klosner· 1 shared Ana FaroiD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersDevelopmental Biology

Florencia CavodeassiORCID

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3 papers

Recent·Most cited

Cachd1 is a novel Frizzled- and LRP6-interacting protein required for neurons to acquire left-right asymmetric character

Gareth T. Powell, Ana Faro, Yuguang Zhao, Heather L. Stickney, Laura Novellasdemunt, Pedro M. Henriques, Gaia Gestri, Esther Redhouse White, Jingshan Ren, Weixian Lu, Rodrigo Young, Thomas A. Hawkins, Florencia Cavodeassi, Quenten Schwarz, Elena Dreosti, , Stephen W. Wilson

This study identifies Cachd1 as a novel Wnt receptor-interacting protein essential for left-right neuronal asymmetry in zebrafish.

bioRxiv · May 16, 2022 · PreprintDevelopmental Biology

The GINS complex is required for the survival of rapidly proliferating retinal and tectal progenitor cells during zebrafish development

Máté Varga, Kitti Csályi, István Bertyák, Dóra K. Menyhárd, Richard J. Poole, Kara L. Cerveny, Dorottya Kövesdi, Balázs Barátki, Hannah Rouse, Zsuzsa Vad, Thomas A. Hawkins, Heather L. Stickney, Florencia Cavodeassi, Quenten Schwarz, Rodrigo Young, Stephen W. Wilson

This study shows that the GINS complex is essential for DNA replication in rapidly dividing retinal and tectal progenitor cells in zebrafish, leading to apoptosis in these cells when GINS function is absent.

bioRxiv · Feb 9, 2020 · PreprintGenetics

Compensatory mechanisms render Tcf7l1a dispensable for eye formation despite its requirement in eye field specification

Rodrigo Young, Thomas A. Hawkins, Florencia Cavodeassi, Heather L. Stickney, Quenten Schwarz, Lisa M. Lawrence, Claudia Wierzbicki, Bowie Yik-Ling Cheng, Jingyuan Luo, Elizabeth Mayela Ambrosio, Allison Klosner, Ian M. Sealy, Jasmine Rowell, Chintan A. Trivedi, Isaac Henry Bianco, , Stephen W. Wilson

The paper shows that Tcf7l1a is essential for eye field specification in zebrafish but not for eye development, as mutants develop normal eyes through compensatory mechanisms.

eLife · Feb 18, 2019Peer-reviewedDevelopmental Biology