Author

Hannah Rouse

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:zebrafish· 2 Research Focus:cancer & tumor microenvironment· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Kara L. CervenyiD· 2 shared Máté VargaiD· 2 shared Stephen W. WilsoniD· 2 shared Anitta R. ChackoiD· 1 shared Balázs Barátki· 1 shared Benjamin O’CallaghaniD· 1 shared Cameron G. RobertsiD· 1 shared Chih-Yao ChungiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersDevelopmental Biology

Kara L. CervenyORCID

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Author profile →

3 papers

Recent·Most cited

Optic nerve innervation promotes Wnt/b-catenin pathway activity and progenitor cell proliferation in the zebrafish optic tectum

Olivia L. Hagen, Yehyun Kim, Elaine E. Kushkowski, Jiahe Yue, Hannah Rouse, Sara Helmstetter, Cameron G. Roberts, Máté Varga, Stephen W. Wilson, Kara L. Cerveny

The paper investigates how optic nerve innervation affects Wnt/b-catenin pathway activity and progenitor cell proliferation in zebrafish optic tectum, showing that innervation promotes survival and proliferation even in darkness.

bioRxiv · Jun 19, 2026 · PreprintDevelopmental Biology

Metabolic remodelling in hiPSC-derived myofibres carrying the m.3243A>G mutation

Gabriel E. Valdebenito, Anitta R. Chacko, Chih-Yao Chung, Preethi Sheshadri, Haoyu Chi, Benjamin O’Callaghan, Monika J. Madej, Henry Houlden, Hannah Rouse, Valle Morales, Katiuscia Bianchi, Francesco Saverio Tedesco, Robert D. S. Pitceathly, Michael R. Duchen

This study generates hiPSC-derived myofibres with the m.3243A>G mutation to model mitochondrial disease, showing metabolic and physiological changes related to mutation load.

Stem Cell Reports · Mar 13, 2025Peer-reviewedCell Biology

The GINS complex is required for the survival of rapidly proliferating retinal and tectal progenitor cells during zebrafish development

Máté Varga, Kitti Csályi, István Bertyák, Dóra K. Menyhárd, Richard J. Poole, Kara L. Cerveny, Dorottya Kövesdi, Balázs Barátki, Hannah Rouse, Zsuzsa Vad, Thomas A. Hawkins, Heather L. Stickney, Florencia Cavodeassi, Quenten Schwarz, Rodrigo Young, Stephen W. Wilson

This study shows that the GINS complex is essential for DNA replication in rapidly dividing retinal and tectal progenitor cells in zebrafish, leading to apoptosis in these cells when GINS function is absent.

bioRxiv · Feb 9, 2020 · PreprintGenetics