Máté Varga

SCAMP quantifies calcification in fish larvae by straightening the notochord and generating standardized mineralization profiles, revealing genotype-specific differences and the effects of inorganic pyrophosphate and methylene blue.

The paper investigates how optic nerve innervation affects Wnt/b-catenin pathway activity and progenitor cell proliferation in zebrafish optic tectum, showing that innervation promotes survival and proliferation even in darkness.

This study compares the behavioral responses of paradise fish and zebrafish in social and non-social tasks, finding that paradise fish show less social bias and more consistent behavior in individual-based assays.

The paper identifies a transposon insertion in the 3UTR of ctnnb2 as the cause of the maternal-effect ich mutation in zebrafish, leading to transcript instability and ventralized embryos.

This study identifies KCNQ3 as a genetic risk factor for fear-related behaviors in dogs and zebrafish, showing gene-environment interactions influence freezing behavior.

The paper catalogs the zebrafish snoRNAome, identifies 68 new snoRNAs, and provides evidence for dynamic and tissue-specific expression patterns.

This paper develops fluorescent probes EvoSiR4 and EvoSiR6 that potently and specifically label CaSR in vitro, in vivo, and ex vivo, outperforming evocalcet and its linker conjugate.

The paper reports the reference genome of the paradise fish (Macropodus opercularis) using long-read sequencing, identifying 20,157 protein-coding genes and demonstrating high completeness.

This study creates and characterizes a zebrafish model for Bloom syndrome, showing that blm mutants exhibit shortened lifespan, reduced fertility, and germ line developmental issues.

This study shows that early-life social isolation in zebrafish alters serotonergic signaling and behavioral responsiveness to challenges, with pharmacological intervention rescuing these effects.

This study shows that the GINS complex is essential for DNA replication in rapidly dividing retinal and tectal progenitor cells in zebrafish, leading to apoptosis in these cells when GINS function is absent.

The paper shows that mutant cells with cell cycle defects either die or differentiate in wild-type environments, while hyperproliferative cells form tumor-like structures and resist differentiation.

SignaFish provides a zebrafish-specific signaling pathway resource built on the SignaLink framework, offering curated and additional interactions for pathway analysis.