Author

Hong Jun Rhee

Max Planck Institute for Multidisciplinary Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:electronic health records· 1

Frequent Co-Authors

Bekir AltasiD· 2 shared Hiroshi KawabeiD· 2 shared JeongSeop RheeiD· 2 shared Manuela SchwarkiD· 2 shared A Ioana WeberiD· 1 shared Albrecht Sigler· 1 shared Anes JuiD· 1 shared Ekaterina V. BorisovaiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersNeuroscience

Bekir AltasORCID

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2 papers

Recent·Most cited

Regulation of excitatory presynaptic activity by Ambra1 protein determines neuronal networks in sex-dimorphic manner

Anes Ju, Bekir Altas, Hong Jun Rhee, Manuela Schwark, Imam A. Hassouna, Albrecht Sigler, Hiroshi Kawabe, Kamal Chowdhury, Hannelore Ehrenreich, Nils Brose, JeongSeop Rhee

This study shows that Ambra1 heterozygosity causes sex-specific reductions in excitatory synaptic activity, leading to E/I imbalance and autism-like behaviors in female mice.

bioRxiv · Dec 19, 2022 · PreprintNeuroscience

The Kaufman oculocerebrofacial syndrome protein Ube3b regulates synapse number by ubiquitinating Ppp3cc.

Mateusz C. Ambrozkiewicz, Ekaterina V. Borisova, Manuela Schwark, Silvia Ripamonti, Theres Schaub, Смородченко Алина Тихоновна, A Ioana Weber, Hong Jun Rhee, Bekir Altas, Rüstem Yilmaz, Susanne G. Mueller, Lars Piepkorn, Stephen T. Horan, Rachel Straussberg, Sami Zaqout, , Hiroshi Kawabe

This study shows that Ube3b regulates dendritic branching and synaptic function by ubiquitinating Ppp3cc, contributing to intellectual disability-like phenotypes in a KOS mouse model.

Molecular Psychiatry · Apr 6, 2020Peer-reviewedNeuroscience