Author

I. Mikicic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:development & morphogenesis· 1 Assay:metagenomics· 1 Research Focus:transcription & RNA processing· 1

Frequent Co-Authors

P. Beli· 3 shared A. Brem· 1 shared A. Plueckthun· 1 shared A. Stier· 1 shared B. Dreier· 1 shared C. Schuelein-Voelk· 1 shared D. Fleischhauer· 1 shared D. J. Mueller· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersCell Biology

P. Beli

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Direct RNA-binding by MYCN mediates feedback from RNA processing to transcription control

D. Papadopoulos, S. A. Ha, D. Fleischhauer, L. Uhl, K. Schneider, I. Mikicic, T. J. Russell, A. Brem, O. R. Valanju, P. Gallant, C. Schuelein-Voelk, H. M. Maric, P. Beli, G. Buechel, S. M. Vos, M. Eilers

This study shows MYCN binds RNA and regulates transcript turnover, with its RNA-binding activity affecting transcription control through competition with DNA-binding.

bioRxiv · Aug 16, 2023 · PreprintCancer Biology

The CRL4B E3 ligase regulates mitosis by recruiting phospho-specific DCAFs

A. Stier, S. Gilberto, W. I. Mohamed, J. Helenius, I. Mikicic, T. Sajic, P. Beli, D. J. Mueller, M. Peter

This study identifies LIS1 and WDR1 as CUL4B-specific DCAFs that bind via phosphorylation, essential for mitosis and brain development, but not the CUL4B-P50L mutant.

bioRxiv · Oct 14, 2022 · PreprintCell Biology

Nuclear myosin VI maintains replication fork stability

J. Shi, K. Hauschulte, I. Mikicic, S. Maharjan, V. Arz, J. B. Heidelberger, J. V. Schaefer, B. Dreier, A. Plueckthun, P. Beli, H. D. Ulrich, H.-P. Wollscheid

This study shows that nuclear myosin VI stabilizes stalled or reversed replication forks by interacting with WRNIP1 to prevent DNA2-mediated degradation during replication stress.

bioRxiv · Jul 28, 2022Cell Biology