Author

J. Kline

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. A. Alizadeh· 1 shared A. A. Mendelson· 1 shared A. Bagaev· 1 shared A. Cooper· 1 shared A. H. Rowe· 1 shared A. Heaston· 1 shared A. R. West· 1 shared A. Saleem· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCancer Biology

A. A. Alizadeh

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Integrative genomic analysis identifies unique immune environments associated with immunotherapy response in diffuse large B cell lymphoma

S. Tumuluru, J. K. Godfrey, A. Cooper, J. Yu, X. Chen, B. W. MacNabb, G. Venkataraman, Y. Zha, B. Pelzer, J. Song, G. Duns, B. J. Sworder, C. Bolen, E. Penuel, E. Postovalova, , J. Kline

This study identifies unique immune environments in DLBCL associated with response to immunotherapy using an integrative multi-omic approach.

bioRxiv · Jan 17, 2024 · PreprintCancer Biology

The mitophagy receptor BNIP3L/Nix coordinates nuclear calcium signaling to modulate the muscle phenotype.

J. T. Field, D. Chapman, Y. Hai, S. Ghavami, A. R. West, B. Ozerklig, A. Saleem, J. Kline, A. A. Mendelson, J. Kindrachuk, B. Triggs-Raine, J. W. Gordon

This study shows Nix regulates muscle phenotype by coordinating mitophagy, ER-phagy, and nuclear calcium signaling, affecting insulin sensitivity and fiber-type composition.

bioRxiv · Mar 18, 2023 · PreprintCell Biology

Identification and characterization of novel proteins from Arizona bark scorpion venom that inhibit Nav1.8, a voltage-gated sodium channel regulator of pain signaling

T. M. Abd El-Aziz, Y. Xiao, J. Kline, H. Gridley, A. Heaston, K. D. Linse, M. J. Ward, D. R. Rokyta, J. D. Stockand, T. R. Cummins, L. Fornelli, A. H. Rowe

The paper identifies four novel scorpion venom proteins that inhibit Nav1.8, a sodium channel involved in pain signaling, through proteomic and electrophysiological analyses.

bioRxiv · Jun 24, 2021Pharmacology & Toxicology