Author

J. Takita

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

T. Tanaka· 2 shared D. Yoshinaga· 1 shared E. Hiejima· 1 shared E. Kume· 1 shared F. Fukumura· 1 shared H. Fujita· 1 shared H. Kubota· 1 shared H. Nihira· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

T. Tanaka

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

HOIL-1L deficiency induces cell cycle alteration which causes immaturity of myocyte and fibrogenesis

K. Akagi, S. Baba, H. Fujita, Y. Fuseya, D. Yoshinaga, H. Kubota, E. Kume, F. Fukumura, K. Matsuda, T. Tanaka, T. Hirata, K. Iwai, J. Takita

This study shows HOIL-1L deficiency leads to cell cycle defects and fibrosis in myotubes and cardiomyocytes, contributing to myopathy and dilated cardiomyopathy.

bioRxiv · Sep 29, 2023 · PreprintCell Biology

Aberrant localization of CDC42 C-terminal variants to the Golgi apparatus drives pyrin inflammasome-dependent autoinflammation

M. Nishitani-Isa, K. Mukai, Y. Honda, H. Nihira, H. Shibata, T. Tanaka, K. Kodama, E. Hiejima, K. Izawa, Y. Kawasaki, M. Osawa, Y. Katata, S. Onodera, T. Watanabe, S. Kure, , T. Yasumi

This study identifies that CDC42 C-terminal variants cause aberrant Golgi localization, leading to pyrin inflammasome activation and autoinflammation.

bioRxiv · Sep 3, 2021Immunology