Author

J. Thomson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:cryo-EM· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1

Frequent Co-Authors

A. J. Dear· 1 shared A. M. Roseman· 1 shared A. R. Godwin· 1 shared C. Baldock· 1 shared C. S. Adamo· 1 shared D. Clow· 1 shared D. F. Holmes· 1 shared G. Meisl· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiophysics

A. J. Dear

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Molecular mechanism of α-synuclein aggregation on lipid membranes revealed.

A. J. Dear, X. Teng, S. R. Ball, J. Lewin, R. I. Horne, D. Clow, N. Harper, K. Yahya, T. C. Michaels, S. Linse, T. P. Knowles, X. Yang, S. C. Brewerton, J. Thomson, J. Habchi, G. Meisl

This study reveals that lipids promote α-synuclein aggregation by enabling elongation, using a kinetic model to describe the co-aggregation process in vitro.

bioRxiv · Oct 20, 2023Biophysics

Fibrillin microfibril structure identifies long-range effects of inherited pathogenic mutations affecting a key regulatory TGFβ-binding site

A. R. Godwin, J. Thomson, D. F. Holmes, C. S. Adamo, G. Sengle, M. J. Sherratt, A. M. Roseman, C. Baldock

The paper reveals the cryo-EM structure of fibrillin microfibrils and shows how mutations disrupt TGFβ signaling by blocking LTBP-1 interaction through long-range structural effects.

bioRxiv · Aug 26, 2022 · PreprintBiochemistry

Age-dependent effects of reduced mTor signalling on life expectancy through distinct physiology

M. J. P. Simons, L. Hartshorne, S. Trooster, J. Thomson, M. Tatar

This study shows that reducing mTor signaling in old age decreases mortality, while early transient reduction has long-lasting benefits, suggesting age-dependent effects of mTor on aging.

bioRxiv · Jul 30, 2019 · PreprintPhysiology