Author

J. Z. Nikolich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

J. L. Uhrlaub· 2 shared A. A. Koshy· 1 shared B. A. Bixby· 1 shared B. J. LaFleur· 1 shared C. Bime· 1 shared C. C. Weinkauf· 1 shared C. M. Bradshaw· 1 shared C. P. Coplen· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

J. L. Uhrlaub

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Chronic Toxoplasma gondii infection curtails the cytotoxic potential of acute T cell responses to West Nile virus in the brain.

J. L. Uhrlaub, K. E. McGovern, E. F. Merritt, M. S. Kuhns, A. A. Koshy, J. Z. Nikolich

The paper shows that chronic Toxoplasma gondii infection reduces the cytotoxic response of T cells to West Nile virus in the brain, increasing susceptibility to WNV.

bioRxiv · Jan 21, 2024 · PreprintImmunology

Characterization of low copy number human angiotensin-convertase 2 (hACE2)-transgenic mice as an improved model of SARS-CoV-2 infection

C. M. Bradshaw, T. Georgieva, T. N. Tankersley, T. Taylor-Doyle, L. Johnson, J. L. Uhrlaub, D. Besselsen, J. Z. Nikolich

This study develops low-copy hACE2 transgenic mice to better model SARS-CoV-2 infection, showing reduced mortality and viral spread compared to high-copy lines.

bioRxiv · Aug 31, 2023 · PreprintImmunology

T-cell cellular stress and reticulocyte signatures, but not loss of naive T lymphocytes, characterize severe COVID-19 in older adults

M. Jergovic, M. Watanabe, R. Bhat, C. P. Coplen, S. A. Sonar, R. Wong, Y. Castaneda, L. Davidson, M. Kala, R. C. Wilson, H. L. Twigg, K. Knox, H. E. Erickson, C. C. Weinkauf, C. Bime, , J. Z. Nikolich

The paper identifies T-cell stress and reticulocyte signatures as markers of severe COVID-19 in older adults, while showing naive T-cell decline is age-related, not disease-specific.

bioRxiv · Dec 21, 2022Immunology