Author

Jinliang Li

Children's NationalPrimary ORCID profile (0000-0002-3018-0570 · 5 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Jinliang LiiD5 papers · Stanford Cardiovascular Institute

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Patricio E. RayiD· 2 shared Zhe HaniD· 2 shared Jharna R. DasiD· 1 shared Jing YuiD· 1 shared Joyce van de LeemputiD· 1 shared Jun‐yi ZhuiD· 1 shared Lian Xu· 1 shared Pankaj Kumar· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

Patricio E. RayORCID

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2 papers

Recent·Most cited

Inflammatory proteolysis generates pathogenic APOL1 fragments with distinct intracellular toxicities in podocytes derived from children with HIV associated nephropathy

Jinliang Li, Ying Yu, Jharna R. Das, Lian Xu, Pankaj Kumar, Zhe Han, Patricio E. Ray

The paper identifies inflammatory proteolysis of APOL1 in podocytes from HIVAN patients, generating toxic fragments with distinct pathogenic functions.

bioRxiv · Aug 17, 2026 · PreprintCell Biology

HIV-1 Nef acts in synergy with APOL1-G1 to induce nephrocyte cell death in a new Drosophila model of HIV-related kidney diseases

Jun‐yi Zhu, Yulong Fu, Joyce van de Leemput, Jing Yu, Jinliang Li, Patricio E. Ray, Zhe Han

This paper introduces a Drosophila model to study HIV-1 Nef and APOL1-G1 interaction, showing they synergistically induce ER stress and nephrocyte cell death.

Disease Models & Mechanisms · Jul 1, 2025Peer-reviewedMolecular Biology