Author

Jonathan P. Richardson

King's College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:metagenomics· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Emily L. PriestiD· 2 shared Julian R. NaglikiD· 2 shared Adrian Lau· 1 shared Aize PellóniD· 1 shared Alexander KempfiD· 1 shared Andrew J. Donkin· 1 shared Andrew J. StaggiD· 1 shared Ann-Kristin KauneiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersMicrobiology

Emily L. PriestORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The IL-1 Family Controls Acute Mucosal Fungal Infection and Mucosal-Systemic Dissemination.

James S. Griffiths, Alexander Kempf, Robert J. Pickering, Emily L. Priest, Olivia K. A. Paulin, Léa Lortal, Andrew J. Donkin, Olivia W. Hepworth, Don N. Wickramasinghe, Aize Pellón, Adrian Lau, Harun Papini, Sarah L. Gaffen, Jonathan P. Richardson, Julian R. Naglik

This study shows the IL-1 family is essential for mucosal defense against Candida albicans, preventing infection and dissemination, but its absence in neutropenic conditions leads to fatal systemic infection.

bioRxiv · Apr 10, 2026 · PreprintMicrobiology

Bacterial suppression of intestinal fungi via activation of human gut γδ T-cells

Liya Mathew, Sean Carlson, Michael Savage, Claire Pardieu, Megan O’Brien, Ellie Crawley, Paul Harrow, Ann-Kristin Kaune, Edward Devlin, Emily L. Priest, Marilena Crescente, Kimberly Martinod, James R. Boot, Marco Gasparetto, Klaartje Bel Kok, , Neil E. McCarthy

The paper shows that bacterial metabolite HMB-PP activates human gut γδ T-cells to suppress fungal growth, but this mechanism is impaired in Crohn's disease.

bioRxiv · Sep 17, 2025 · PreprintImmunology