Author

Jyllica Aurelio

Moffitt Cancer Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Ahmed RamadaniD· 1 shared Andriy MarusykiD· 1 shared Ben CreelaniD· 1 shared Bina DesaiiD· 1 shared Bruna PelliniiD· 1 shared Daniel Abate‐DagaiD· 1 shared David BasantaiD· 1 shared Elena Martinez PlanesiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

Ahmed RamadanORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Dual stimulation of CD40 and 41BB pathways during ex-vivo TIL expansion enhances CD8+ T cell expansion

R. A. M. Rossetti, Matthew S. Beatty, Junior Cianne, Johannes R. Ali, Kylie Harris, Ahmed Ramadan, Michael Grant, Elena Martinez Planes, Jyllica Aurelio, Lilit Karapetyan, Ben Creelan, Shari Pilon‐Thomas, Patrick Hwu, Vincent C. Luca, Daniel Abate‐Daga

This study shows that dual stimulation of CD40 and 41BB pathways using a bi-specific molecule enhances CD8+ T cell expansion in TIL cultures.

bioRxiv · Jul 26, 2026 · PreprintImmunology

Pharmacokinetic Acceleration via CYP3A4 Hyperactivation as a Clinically Actionable Mechanism of Targeted Therapy Resistance in NSCLC

Pragya Kumar, Menkara Henry, Julia Pelesko, Bina Desai, Malgorzata Tyczynska Weh, Ramu Kakumanu, Min Liu, Natália Souza Nunes Siqueira, Robert Vander Velde, Viktoriya Marusyk, Jyllica Aurelio, Eric B. Haura, Jhanelle Gray, Bruna Pellini, David Basanta, Andriy Marusyk

This paper identifies CYP3A4 hyperactivation as a tumor-extrinsic resistance mechanism in NSCLC, showing it reduces drug exposure and leads to resistance, and proposes dosing optimization and CYP3A4 inhibition as solutions.

bioRxiv · May 20, 2026 · PreprintCancer Biology