Author

K. Etori

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

H. Nakajima· 2 shared K. Hattori· 2 shared K. Ikeda· 2 shared K. Yokote· 2 shared S. Tanaka· 2 shared T. Kasuya· 2 shared A. Suto· 1 shared C. Betsholtz· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersPathology

H. Nakajima

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2 papers

Recent·Most cited

Semaphorin 3G exacerbates joint inflammation through the accumulation and proliferation of macrophages in the synovium

J. Shoda, S. Tanaka, K. Etori, K. Hattori, T. Kasuya, K. Ikeda, Y. Maezawa, A. Suto, K. Suzuki, J. Nakamura, Y. Maezawa, M. Takemoto, C. Betsholtz, K. Yokote, S. Ohtori, H. Nakajima

This study identifies Semaphorin 3G as a factor that worsens joint inflammation by promoting macrophage accumulation and proliferation in RA synovium.

bioRxiv · Feb 12, 2022Pathology

Immunological features that determine the strength of antibody responses to BNT162b2 mRNA vaccine against SARS-CoV-2

T. Kageyama, S. Tanaka, K. Etori, K. Hattori, K. Miyachi, T. Kasuya, T. Iwamoto, K. Ikeda, H. Igari, K. Yokote, H. Nakajima

The paper identifies immunological features, including B cell frequencies and CD8+ T cell activation, that correlate with antibody response strength to the BNT162b2 mRNA vaccine.

bioRxiv · Jun 21, 2021Immunology