Author

K. Ronacher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 3 Microbiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 5 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 4 Research Focus:cell type annotation· 3 Assay:proteomics· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Assay:clinical cohort / trial· 1

Frequent Co-Authors

H. Bielefeldt-Ohmann· 4 shared M. D. Ngo· 4 shared S. Bartlett· 4 shared C. X. Foo· 3 shared M. M. Rosenkilde· 3 shared R. Sinha· 3 shared B. J. Arachchige· 2 shared K. Short· 2 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersImmunology

H. Bielefeldt-Ohmann

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Cardiovascular symptoms of PASC are associated with trace-level cytokines that affect the function of human pluripotent stem cell derived cardiomyocytes

J. Sinclair, C. Vedelago, F. Ryan, M. Carney, M. Redd, M. Lynn, B. Grubor-Bauk, Y. Cao, A. Henders, K. Y. Chew, D. Gilroy, K. Greaves, L. Labzin, L. Ziser, K. Ronacher, , K. Short

This study identifies trace-level cytokines in PASC patients linked to cardiovascular symptoms and shows their impact on cardiomyocyte function using nanotechnology detection.

bioRxiv · Apr 11, 2024 · PreprintMicrobiology

Oxysterols drive inflammation via GPR183 during influenza virus and SARS-CoV-2 infection

C. X. Foo, S. Bartlett, K. Y. Chew, M. D. Ngo, H. Bielefeldt-Ohmann, B. J. Arachchige, B. Matthews, S. Reed, R. Wang, M. J. Sweet, L. Burr, J. E. Sinclair, R. Parry, A. Khromykh, K. R. Short, , K. Ronacher

This study identifies oxysterols and GPR183 as drivers of inflammation in the lung during influenza and SARS-CoV-2 infections, showing that targeting GPR183 reduces inflammation and disease severity.

bioRxiv · Jun 14, 2022Immunology

A blunted GPR183/oxysterol axis during dysglycemia results in delayed recruitment of macrophages to the lung during M. tuberculosis infection

M. D. Ngo, S. Bartlett, H. Bielefeldt-Ohmann, C. X. Foo, R. Sinha, B. J. Arachchige, S. J. Reed, T. Mandrup-Poulsen, M. M. Rosenkilde, K. Ronacher

This study shows that dysglycemia impairs the GPR183/oxysterol axis, leading to delayed macrophage recruitment and worsened tuberculosis outcomes in mice.

bioRxiv · Jan 5, 2022Immunology

Pre-diabetes increases tuberculosis disease severity, while high body fat without impaired glucose tolerance is protective

R. Sinha, M. D. Ngo, S. Bartlett, H. Bielefeldt-Ohmann, S. Keshvari, S. Z. Hasnain, M. L. Donovan, J. C. Kling, A. Blumenthal, C. Chen, K. Short, K. Ronacher

bioRxiv · Apr 6, 2021Immunology

GPR183 regulates interferons and bacterial growth during Mycobacterium tuberculosis infection: interaction with type 2 diabetes and TB disease severity

S. Bartlett, A. T. Gemiarto, M. D. Ngo, H. Sajiir, S. Hailu, R. Sinha, C. X. Foo, L. Kleynhans, H. Tshivhula, T. Webber, H. Bielefeldt-Ohmann, N. P. West, A. M. Hiemstra, C. E. MacDonald, L. von Voss Christensen, , K. Ronacher

bioRxiv · Jul 15, 2020Microbiology