Author

L. A. Campbell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. A. Brown· 1 shared B. Z. Stanger· 1 shared C. M. Johnson· 1 shared E. Stelow· 1 shared J. R. Pitarresi· 1 shared K. E. Lee· 1 shared K. GBEDANDE· 1 shared K. Leong· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

B. A. Brown

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

A histone methylation-MAPK signaling axis drives durable epithelial-mesenchymal transition in hypoxic pancreas cancer

B. A. Brown, P. J. Myers, S. J. Adair, J. R. Pitarresi, S. K. Sah-Teli, L. A. Campbell, W. S. Hart, M. Barbeau, K. Leong, N. Seyler, W. Kane, K. E. Lee, E. Stelow, M. Jones, M. C. Simon, , M. J. Lazzara

This study identifies a hypoxia-driven histone methylation-MAPK feedback loop that sustains EMT in pancreatic cancer, with potential therapeutic implications.

bioRxiv · Oct 19, 2022 · PreprintCancer Biology

Functionally competent CD4+ T cells express high levels of T-bet in Plasmodium chabaudi infected young mice

M. R. Smith, K. GBEDANDE, C. M. Johnson, L. A. Campbell, L. B. Richard, R. S. Onjiko, N. Domingos, M. M. Opata

The paper shows that Plasmodium chabaudi infection in young mice leads to increased T-bet expression in CD4+ T cells, suggesting enhanced Th1 commitment and potential cerebral symptoms.

bioRxiv · Feb 8, 2022 · PreprintImmunology