Author

L. H. Ibrahim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

M. J. Bollong· 3 shared A. B. Chan· 2 shared K. A. Lamia· 2 shared A. Stolz· 1 shared A. Ta· 1 shared A. Verhelle· 1 shared B. J. Altman· 1 shared C. A. Cuoco· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersCell Biology

M. J. Bollong

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

An mTOR-independent Macroautophagy Activator Ameliorates Tauopathy and Prionopathy Neurodegeneration Phenotypes

L. Yoon, R. C. Botham, A. Verhelle, Y. Wu, C. Cole, E. P. Tan, L. A. Massey, P. Sanz-Martinez, C. C. Chou, J. Xu, L. P. Elia, K. Lee, S. R. Labra, G. M. Kline, Q. Xiao, , S. Encalada

The paper identifies CCT020312, an mTOR-independent autophagy activator, which reduces tauopathy and prionopathy neurodegeneration phenotypes by enhancing autophagy flux and clearing protein aggregates.

bioRxiv · Sep 29, 2022 · PreprintCell Biology

Circadian disruption enhances HSF1 signaling and tumorigenesis in Kras-driven lung cancer

M. Pariollaud, L. H. Ibrahim, E. Irizarry, R. M. Mello, A. B. Chan, B. J. Altman, R. J. Shaw, M. J. Bollong, L. Wiseman, K. A. Lamia

This study shows that chronic circadian disruption increases tumor burden in KRAS-driven lung cancer by enhancing HSF1 signaling and tumorigenesis.

bioRxiv · Jan 31, 2022Cell Biology

CRY2 missense mutations suppress P53 and enhance cell growth

A. B. Chan, G. C. G. Parico, J. L. Fribourgh, L. H. Ibrahim, M. J. Bollong, C. L. Partch, K. A. Lamia

The paper identifies CRY2 mutations that suppress P53 and enhance cell growth by disrupting interactions with SCFFBXL3 and affecting circadian rhythms.

bioRxiv · Jan 8, 2021Cell Biology