Author

M. B. Ferretti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

D. C. Schultz· 1 shared E. R. Worden-Sapper· 1 shared J. Hulahan· 1 shared J. M. Burke· 1 shared J. Miller· 1 shared J. S. Lee· 1 shared K. Ayyanathan· 1 shared K. W. Lynch· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMicrobiology

D. C. Schultz

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Evolutionary arms race between SARS-CoV-2 and interferon signaling via dynamic interaction with autophagy.

J. S. Lee, M. Dittmar, J. Miller, M. Li, K. Ayyanathan, M. B. Ferretti, J. Hulahan, K. Whig, Z. Etwebi, T. Griesman, D. C. Schultz, S. Cherry

This study identifies how SARS-CoV-2 evolves to counteract host interferon signaling through interactions with autophagy pathways.

bioRxiv · Nov 13, 2023 · PreprintMicrobiology

RNase L-mediated RNA decay alters 3' end formation and splicing of host mRNAs

J. M. Burke, N. Ripin, M. B. Ferretti, L. A. St Clair, E. R. Worden-Sapper, S. L. Sawyer, R. Perera, K. W. Lynch, R. Parker

This study shows that RNase L-mediated RNA decay causes RNA-binding proteins to shift from cytoplasm to nucleus, altering splicing and 3' end formation of host mRNAs, especially interferons.

bioRxiv · Jan 28, 2022 · PreprintImmunology