Author

M. D. Newton

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Organism:rat· 1

Frequent Co-Authors

A. Kaczmarczyk· 1 shared C. Barroso· 1 shared D. S. Rueda· 1 shared E. Maeots· 1 shared E. Martinez-Perez· 1 shared K. C. Baker· 1 shared K. Gawronski· 1 shared L. Junginger· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiophysics

A. Kaczmarczyk

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2 papers

Recent·Most cited

Single-molecule analysis reveals cooperative stimulation of Rad51 filament nucleation and growth by mediator proteins

O. Belan, C. Barroso, A. Kaczmarczyk, R. Anand, S. Federico, N. O'Reilly, M. D. Newton, E. Maeots, R. I. Enchev, E. Martinez-Perez, D. S. Rueda, S. J. Boulton

The paper uses single-molecule imaging to show how BRC-2 and RFS-1/RIP-1 mediate RAD-51 filament nucleation and growth during homologous recombination.

bioRxiv · Nov 5, 2020Biophysics

Traumatic Joint Injury Induces Acute Catabolic Bone Turnover Concurrent with Articular Cartilage Damage in a Rat Model of Post-Traumatic Osteoarthritis

T. Maerz, M. D. Newton, M. Fleischer, S. E. Hartner, K. Gawronski, L. Junginger, K. C. Baker

This study shows that traumatic joint injury in rats causes acute bone catabolism and cartilage damage, with reduced bone formation and increased osteoclast activity.

bioRxiv · Apr 10, 2020Physiology