Author

M. Deleidi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Armento· 1 shared A. Jacob· 1 shared A. Viejo-Borbolla· 1 shared B. Ritter· 1 shared E. Kilger· 1 shared F. J. Zapatero-Belinchon· 1 shared F. Wegner· 1 shared G. Gerold· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Armento

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Innate immune response to SARS-1 CoV-2 infection contributes to neuronal damage in human iPSC-derived peripheral neurons

V. Passos, L. M. Henkel, J. Wang, F. J. Zapatero-Belinchon, R. Moeller, G. Sun, I. Waltl, B. Ritter, K. A. Kropp, S. Zhu, M. Deleidi, U. Kalinke, G. Hoeglinger, G. Gerold, F. Wegner, A. Viejo-Borbolla

This study infects human iPSC-derived CNS and PNS neurons with SARS-CoV-2 and finds that PNS neurons experience more severe neurodegeneration linked to innate immune activation.

bioRxiv · Nov 18, 2022Microbiology

Loss of Complement Factor H impairs antioxidant capacity and energy metabolism of human RPE cells

A. Armento, S. Honisch, V. Panagiotakopoulou, I. Sonntag, A. Jacob, E. Kilger, M. Deleidi, S. J. Clark, M. Ueffing

This study shows that loss of Complement Factor H in human RPE cells reduces antioxidant capacity and impairs energy metabolism, contributing to AMD pathogenesis.

bioRxiv · Jan 8, 2020Cell Biology