Author

M. Gharaee-Kermani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

F. Ma· 2 shared G. A. Hile· 2 shared J. E. Gudjonsson· 2 shared J. M. Kahlenberg· 2 shared A. Bigger-Allen· 1 shared A. C. Billi· 1 shared A. Hashemi Gheinani· 1 shared A. M. Victory· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersImmunology

F. Ma

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Epidermal ZBP1 stabilizes mitochondrial Z-DNA to drive UV-induced IFN signaling in autoimmune photosensitivity

B. Klein, M. B. Reynolds, B. Xu, M. Gharaee-Kermani, Y. Gao, C. C. Berthier, S. Henning, S. N. Loftus, K. E. McNeely, A. M. Victory, C. J. Dobry, G. A. Hile, F. Ma, J. L. Turnier, J. E. Gudjonsson, , J. M. Kahlenberg

The paper identifies ZBP1 and mitochondrial Z-DNA as drivers of autoimmune photosensitivity through cGAS-STING activation in response to UVB.

Science Immunology · Mar 7, 2025Peer-reviewedImmunology

Integrated omics analysis unveils a DNA damage response to neurogenic injury

A. Hashemi Gheinani, B. S. Sack, A. Bigger-Allen, H. Thaker, H. Atta, G. Lambrinos, K. Costa, C. Doyle, M. Gharaee-Kermani, S. Patalano, M. Piper, J. F. Cotellessa, D. Vitko, H. Li, M. Kadayil Prabhakaran, , R. M. Adam

The paper uses multi-omics analysis to study molecular changes in the bladder after spinal cord injury in rats and identifies DNA damage response pathways modulated by inosine treatment.

bioRxiv · Dec 10, 2023 · PreprintCell Biology

Non-lesional and Lesional Lupus Skin Share Inflammatory Phenotypes that Drive Activation of CD16+ Dendritic Cells

A. C. Billi, F. Ma, O. Plazyo, M. Gharaee-Kermani, R. Wasikowski, G. A. Hile, X. Xing, C. M. Yee, S. M. Rizvi, M. P. Maz, F. Wen, L. C. Tsoi, M. Pellegrini, R. L. Modlin, J. E. Gudjonsson, J. M. Kahlenberg

The paper compares non-lesional and lesional lupus skin to show they share inflammatory features that activate CD16+ dendritic cells.

bioRxiv · Sep 17, 2021 · PreprintImmunology