Author

M. H. Katz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:human· 4 Research Focus:cell type annotation· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:clustering· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Assay:scRNA-seq· 2

Frequent Co-Authors

A. Maitra· 2 shared A. Semaan· 2 shared B. M. Stephens· 2 shared M. P. Kim· 2 shared P. A. Guerrero· 2 shared V. Bernard· 2 shared A. D. Singhi· 1 shared A. Ngo-Huang· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Maitra

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Aerobic exercise promotes PDAC vascular normalization through S1PR1 signaling in tumor endothelial cells

R. Ballaro, S. Pareek, J. Lee, H. Patel, P. Tirumala, G. Liu, K. Hayek, H. Savage, B. Guerrouahen, A. Ngo-Huang, N. Parker, M. H. Katz, M. Petzel, F. McAllister, R. Wright, , K. Schadler

This study shows aerobic exercise improves PDAC vascular function and chemotherapy efficacy via S1PR1 signaling in tumor endothelial cells.

bioRxiv · Feb 14, 2024 · PreprintCancer Biology

Occult polyclonality of preclinical pancreatic cancer models drives in vitro evolution.

M. E. Monberg, H. Geiger, R. Sharma, J. J. Lee, A. Semaan, V. Bernard, D. B. Swartzlander, B. M. Stephens, K. Chen, M. H. Katz, N. Robine, P. A. Guerrero, A. Maitra

This study reveals high intratumoral heterogeneity in pancreatic cancer cell lines and highlights issues with reproducibility and validity of preclinical models.

bioRxiv · Apr 13, 2021Cancer Biology

DDR1-INDUCED NEUTROPHIL EXTRACELLULAR TRAPS DRIVE PANCREATIC CANCER METASTASIS

J. Deng, Y. Kang, C.-C. Cheng, X. Li, B. Dai, M. H. Katz, T. Men, M. P. Kim, E. Koay, H. Huang, R. A. Brekken, J. B. Fleming

This study shows DDR1 signaling in pancreatic cancer cells promotes metastasis by inducing CXCL5 production, which recruits neutrophils to form extracellular traps that facilitate cancer cell invasion.

bioRxiv · Aug 5, 2020Cancer Biology

Single Cell Transcriptomics of Pancreatic Cancer Precursors Demonstrates Epithelial and Microenvironmental Heterogeneity as an Early Event in Neoplastic Progression

V. Bernard, A. Semaan, J. Huang, F. A. San Lucas, F. C. Mulu, B. M. Stephens, P. A. Guerrero, Y. Huang, J. Zhao, N. Kamyabi, S. Sen, P. A. Scheet, C. M. Taniguchi, M. P. Kim, C.-W. Tzeng, , H. A. Alvarez

This study uses single-cell RNA sequencing to analyze transcriptomic changes in pancreatic cancer precursors, revealing epithelial and microenvironmental heterogeneity during progression to cancer.

bioRxiv · Apr 26, 2018Pathology