Author

M. H. Koster

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1

Frequent Co-Authors

A. de Bruin· 3 shared A. B. Kohan· 1 shared A. Damaskou· 1 shared A. E. Tijhuis· 1 shared A. F. Svendsen· 1 shared A. G. Groenen· 1 shared A. Gerding· 1 shared A. Jurdzinski· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

A. de Bruin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

T cell cholesterol efflux suppresses apoptosis and senescence and increases atherosclerosis in middle aged mice

V. Bazioti, A. M. La Rose, S. Maassen, F. Bianchi, R. de Boer, E. Guilbaud, A. F. Svendsen, A. G. Groenen, A. Marmolejo-Garza, M. H. Koster, N. J. Kloosterhuis, A. T. Pranger, M. Langelaar-Makkinje, A. de Bruin, B. van de Sluis, , M. Westerterp

This study shows that T-cell cholesterol efflux via ABCA1/ABCG1 suppresses apoptosis and senescence, but paradoxically reduces atherosclerosis in middle-aged mice.

bioRxiv · Apr 10, 2022Immunology

Cancer tolerance to chromosomal instability is driven by Stat1 inactivation in vivo

M. Schubert, C. Hong, L. J. Jilderda, M. Requesens-Rueda, A. E. Tijhuis, J. E. Simon, P. L. Bakker, J. Cooper, A. Damaskou, R. Wardenaar, B. Bakker, S. Gupta, A. van den Brink, L. Andrade-Ruiz, M. H. Koster, , F. Foijer

The paper identifies Stat1 inactivation as a mechanism by which aneuploid cancers evade immune surveillance in mice and humans.

bioRxiv · Dec 3, 2021 · PreprintCancer Biology

Spontaneous liver disease in wild-type C57BL/6JOlaHsd mice fed semisynthetic diet

O. A. H. O. Ronda, B. J. M. van de Heijning, A. de Bruin, R. E. Thomas, I. Martini, M. Koehorst, A. Gerding, M. H. Koster, V. W. Bloks, A. Jurdzinski, N. L. Mulder, R. Havinga, E. M. van der Beek, D.-J. Reijngoud, F. Kuipers, H. J. Verkade

This study identifies a spontaneous liver disease in C57BL/6JOlaHsd mice fed a semisynthetic diet, characterized by bimodal liver weight distribution and metabolic abnormalities.

bioRxiv · Apr 8, 2020Pathology