Author

M. Kaileh

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Singh· 3 shared R. Sen· 3 shared A. Binkercosen· 1 shared A. L. Roy· 1 shared A. Roy· 1 shared A. Verma· 1 shared A. Weiner· 1 shared B. Long· 1 shared

Activity by Year

2019-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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3 shared papersCell Biology

A. Singh

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cardiomyocyte-specific adenylyl cyclase type-8 overexpression induces activation of RelA together with myocardial and systemic inflammation

V. Kumar, K. C. Bermea, D. Kumar, A. Singh, A. Verma, M. Kaileh, R. Sen, E. G. Lakatta, L. Adamo

This study shows that overexpressing adenylyl cyclase type-8 in cardiomyocytes activates RelA and causes myocardial and systemic inflammation, leading to accelerated aging and reduced lifespan.

bioRxiv · Jul 15, 2023 · PreprintCell Biology

Morphomechanic tuning of ERK by actin-TFII-IΔ regulates cell identity

Q. Wu, J. Zhang, B. Long, X. Hu, B. Mafra de Faria, S. M. Scalf, K. Karatepe, W. Cao, N. Tsopoulidis, A. Binkercosen, M. Yagi, A. Weiner, M. Kaileh, E. M. De La Cruz, A. L. Roy, , S. Guo

The paper shows that actin-TFII-IΔ modulates ERK activity through cell height to direct cell fate during reprogramming.

bioRxiv · Jun 2, 2023 · PreprintCell Biology

Transcription factor TFII-I fine tunes innate properties of B lymphocytes

A. Singh, M. Kaileh, S. De, K. Mazan-Mamczarz, D. Bayarsaihan, R. Sen, A. Roy

This study shows that TFII-I is essential for maintaining innate B cell properties, with its loss causing myeloid-like transcriptomic changes and increased IgG3 switching.

Frontiers in Immunology · Jan 23, 2023Peer-reviewedImmunology

Mitogen-independent cell cycle progression in B lymphocytes

A. Singh, M. H. Spitzer, J. P. Joy, M. Kaileh, X. Qiu, G. P. Nolan, R. Sen

This study shows B lymphocytes can undergo multiple mitosis-independent divisions after the first mitosis, driven by unique G1 phase characteristics and limited by apoptosis, with Birc5 playing an essential role.

bioRxiv · Oct 7, 2019Cell Biology