Author

M. M. Cho

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. E. Quamine· 2 shared C. M. Capitini· 2 shared D. P. Turicek· 2 shared A. Biju· 1 shared A. K. Erbe· 1 shared A. Leduc· 1 shared A. Naji· 1 shared A. P. Montalvo· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. E. Quamine

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

DEC1 is an adult clock regulator that links circadian rhythms to functional β-cell maturation

Z. Gao, M. Liu, A. P. Montalvo, Z. L. Gruskin, A. Leduc, Y. Xie, S. Preza, J. Du, X. Liu, V. Huhn, Z. Cheng, C. Krapp, M. Haemmerle, A. V. Rozo, X. Wen, , J. R. Alvarez-Dominguez

This study shows DEC1 regulates circadian rhythms in β-cells, linking them to metabolic maturation and glucose responsiveness.

bioRxiv · Aug 11, 2023 · PreprintCell Biology

CD155 blockade enhances allogeneic natural killer cell-mediated antitumor response against osteosarcoma

M. M. Cho, L. Song, A. E. Quamine, F. Szewc, L. Shi, J. D. Ebben, D. P. Turicek, J. M. Kline, D. M. Burpee, E. O. Lafeber, M. F. Phillips, A. S. Ceas, A. K. Erbe, C. M. Capitini

This study shows CD155 blockade with allogeneic NK cell infusion improves survival against osteosarcoma in mice, with benefits mediated through DNAM-1.

bioRxiv · Jun 7, 2023Cancer Biology

CXCR4 allows T cell acute lymphoblastic leukemia to escape from JAK1/2 and BCL2 inhibition through CNS infiltration

K. L. Walker, S. P. Rinella, N. J. Hess, D. P. Turicek, S. A. Kabakov, F. Zhu, M. N. Bouchlaka, S. L. Olson, M. M. Cho, A. E. Quamine, A. S. Feils, T. B. Gavcovich, L. Rui, C. M. Capitini

This study shows that combining JAK1/2 and BCL2 inhibitors in T-ALL leads to CNS infiltration, which is reduced by adding a CXCR4 inhibitor.

bioRxiv · Mar 6, 2021Cancer Biology