Author

M. Pulina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

A. Bispo· 1 shared A. Gopalan· 1 shared A. Jedlicki· 1 shared A. Lam· 1 shared A. Sinha· 1 shared C. Mao· 1 shared C. Sawyers· 1 shared D. Pe'er· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPharmacology & Toxicology

A. Bispo

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2 papers

Recent·Most cited

The neuroendocrine transition in prostate cancer is dynamic and dependent on ASCL1

R. Romero, T. Chu, T. J. Gonzalez-Robles, P. Smith, Y. Xie, H. Kaur, S. Yoder, H. Zhao, C. Mao, W. Kang, M. Pulina, K. E. Lawrence, A. Gopalan, S. Zaidi, K. Yoo, , C. Sawyers

This study identifies ASCL1 as a critical driver of neuroendocrine lineage transition in prostate cancer using an in vivo model and spatial transcriptomics.

bioRxiv · Apr 9, 2024Cancer Biology

Rational development of a small-molecule activator of CK1γ2 that decreases C99 and beta-amyloid levels

V. H. Bustos, Y. K. Sunkari, A. Sinha, M. Pulina, A. Bispo, M. Hopkins, A. Lam, S. F. Kriegsman, E. Mui, E. Chang, A. Jedlicki, H. Rosenthal, M. Flajolet, P. Greengard, S. Sinha

This study identifies CK1γ2 as a therapeutic target for Alzheimer's disease by showing its activation reduces C99 and beta-amyloid levels through increased PS1-Ser367 phosphorylation.

bioRxiv · Apr 28, 2023Pharmacology & Toxicology