Author

M. Seiki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Kobayashi· 1 shared J. Gohda· 1 shared J.-i. Inoue· 1 shared K. Semba· 1 shared K. Tomita· 1 shared K. Tsumoto· 1 shared M. Hoshino· 1 shared M. Yamamoto· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

A. Kobayashi

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2 papers

Recent·Most cited

Metalloproteinase-dependent and TMPRSS2-independnt cell surface entry pathway of SARS-CoV-2 requires the furin-cleavage site and the S2 domain of spike protein

M. Yamamoto, J. Gohda, A. Kobayashi, K. Tomita, Y. Hirayama, N. Koshikawa, M. Seiki, K. Semba, T. Akiyama, Y. Kawaguchi, J.-i. Inoue

This study identifies a TMPRSS2-independent metalloproteinase-dependent entry pathway for SARS-CoV-2 that requires the furin cleavage site and S2 domain of the spike protein, highlighting new therapeutic targets.

bioRxiv · Dec 14, 2021 · PreprintMicrobiology

Binding mechanism underlying FIH-1 suppression caused by the N-terminal disordered region of Mint3

T. Ten, S. Nagatoishi, M. Hoshino, Y. Nakayama, M. Seiki, T. Sakamoto, K. Tsumoto

This study investigates the binding mechanism between Mint3 and FIH-1, identifying a novel core site and thermodynamic contributions of Mint3's disordered region.

bioRxiv · May 10, 2021Biochemistry