Author

M. V. Surve

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. Lin· 2 shared J. Rajagopal· 2 shared A. Anil· 1 shared A. Banerjee· 1 shared A. Beckett· 1 shared A. E. Green· 1 shared A. J. Crossen· 1 shared A. M. Tsankov· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

B. Lin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A deep lung cell atlas reveals cytokine-mediated lineage switching of a rare cell progenitor of the human airway epithelium

A. Waghray, I. Monga, B. Lin, V. Shah, M. Slyper, B. Giotti, J. Xu, J. Waldman, D. Dionne, L. T. Nguyen, W. Lou, P. Cai, E. Park, C. Muus, J. Sun, , J. Rajagopal

This study identifies a rare progenitor cell population in the human airway that can differentiate into ionocytes or tuft cells, and shows cytokines can shift their lineage towards tuft cells in asthma and CF.

bioRxiv · Nov 28, 2023 · PreprintCell Biology

Single Cell Transcriptomes, Lineage, and Differentiation of Functional Airway Microfold Cells

M. V. Surve, B. Lin, J. L. Reedy, A. J. Crossen, A. Xu, B. S. Klein, J. M. Vyas, J. Rajagopal

This study identifies and characterizes airway Microfold (M) cells using single-cell transcriptomics, revealing their lineage, differentiation pathways, and functional role in antigen sampling via CR2-dependent endocytosis.

bioRxiv · Aug 6, 2023Cell Biology

Structural insights into loss of function of a pore forming toxin and its role in pneumococcal adaptation to an intracellular lifestyle

D. C. Badgujar, A. Anil, A. E. Green, M. V. Surve, S. Madhavan, A. Beckett, I. Prior, B. K. Godsora, S. B. Patil, P. K. More, S. Guha Sarkar, A. Mitchell, R. Banerjee, P. P. Phale, T. J. Mitchell, , A. Banerjee

This paper identifies structural changes in a non-hemolytic pneumolysin variant that disable pore formation, enabling pneumococcus to adopt an intracellular lifestyle with reduced inflammation and increased host tolerance.

PLOS Pathogens · Nov 20, 2020Peer-reviewedMicrobiology