Author

M. Vermeer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

B. Becher· 2 shared M. van den Broek· 2 shared S. Tugues· 2 shared A. J. H. Suurmeijer· 1 shared A. Kalmykov· 1 shared A. Lee· 1 shared A. Moor· 1 shared A. S. Teplenin· 1 shared

Activity by Year

2020-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

B. Becher

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Conventional NK cells and tissue-resident ILC1s join forces to control liver metastasis

L. Ducimetiere, G. Lucchiari, G. Litscher, M. Nater, L. Heeb, N. G. Nunez, L. Wyss, D. Burri, M. Vermeer, J. Gschwend, A. Moor, B. Becher, M. van den Broek, S. Tugues

This study identifies the non-redundant roles of conventional NK cells and tissue-resident ILC1s in controlling liver metastasis through distinct functions in seeding and outgrowth.

bioRxiv · Jul 17, 2020Immunology

CD39+PD-1+CD8+ T cells mediate metastatic dormancy in breast cancer

P. Tallon de Lara, H. Castanon, M. Vermeer, N. Nunez, K. Silina, B. Sobottka, J. Urdinez, V. Cecconi, F. Movahedian Attar, S. Hiltbrunner, I. Glarner, H. Moch, S. Tugues, B. Becher, M. van den Broek

This study identifies CD39+PD-1+CD8+ T cells as critical for metastatic dormancy in breast cancer, showing their role in suppressing tumor growth and their potential as a biomarker and immunotherapy target.

bioRxiv · Jun 2, 2020Cancer Biology

Dynamic Loading of Human Engineered Heart Tissue Enhances Contractile Function and Drives Desmosome-linked Disease Phenotype

J. M. Bliley, M. Vermeer, R. M. Duffy, I. Batalov, D. Kramer, J. W. Tashman, D. J. Shiwarski, A. Lee, A. S. Teplenin, L. Volkers, B. Coffin, M. F. Hoes, A. Kalmykov, R. N. Palchesko, Y. Sun, , A. W. Feinberg

The paper develops a dynamic EHT model to enhance contractile function and study desmosome-linked disease phenotypes in engineered heart tissues.

bioRxiv · May 25, 2020Bioengineering