Author

M. Viggars

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Organism:human· 1

Frequent Co-Authors

D. J. Owens· 2 shared A. Brull· 1 shared A. Ferry· 1 shared A. P. Sharples· 1 shared C. Coirault· 1 shared C. E. Stewart· 1 shared D. C. Hughes· 1 shared D. C. Turner· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

D. J. Owens

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2 papers

Recent·Most cited

Lamin-related congenital muscular dystrophy alters mechanical signaling and skeletal muscle growth

D. J. Owens, J. Messeant, S. Moog, M. Viggars, A. Ferry, K. Mamchaoui, E. Lacene, N. Romero, A. Brull, G. Bonne, G. S. Butler-Bowne, C. Coirault

This study investigates how LMNA mutations affect skeletal muscle growth and mechanical signaling in muscle stem cells and mouse models, finding impaired differentiation and hypertrophy in LMNA-CMD.

bioRxiv · Aug 6, 2020Physiology

Knockdown of the E3 Ubiquitin ligase UBR5 and its role in skeletal muscle anabolism

D. C. Turner, D. C. Hughes, L. M. Baehr, R. A. Seaborne, M. Viggars, J. C. Jarvis, P. P. Gorski, C. E. Stewart, D. J. Owens, S. C. Bodine, A. P. Sharples

This study shows that knocking down UBR5 leads to skeletal muscle atrophy by disrupting anabolic signaling pathways.

bioRxiv · Jun 5, 2020Cell Biology