Author

Mallikarjun Patil

University of Alabama at BirminghamPrimary ORCID profile (0000-0002-9238-7657 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Mallikarjun PatiliD1 paper · Nizam's Institute of Medical Sciences

Biology Disciplines

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Biochemistry· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Alek D. PeterliniD· 1 shared Alexandre ProlaiD· 1 shared Allison M. ManueliD· 1 shared Annelise M. PossiD· 1 shared Ashleigh D. Whatley· 1 shared Ashley S. HarmsiD· 1 shared Chase Bolding· 1 shared Christopher A. RisleyiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperBiochemistry

Alek D. PeterlinORCID

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Author profile →

2 papers

Recent·Most cited

High-Dimensional Single-Cell Analysis Reveals Coordinated Age-Dependent Neuroinflammatory Microglia-T cell Circuits in the Brain

Md Akkas Ali, Md Hasanul Banna Siam, Donald Vardaman, Chase Bolding, J. Nicholas Brazell, Ashleigh D. Whatley, Christopher A. Risley, Harrison Tidwell, Syed Nakib Hossain, Juhi Samal, Ashley S. Harms, Mallikarjun Patil, Daniel J. Tyrrell

This study identifies age-related neuroinflammatory microglia-T cell circuits in a mouse model of vascular dementia using multi-modal high-dimensional analysis.

bioRxiv · Dec 13, 2025 · PreprintImmunology

Cardiolipin deficiency disrupts electron transport chain and drives steatohepatitis

Marisa J. Brothwell, Guoshen Cao, J. Alan Maschek, Annelise M. Poss, Alek D. Peterlin, Liping Wang, Talia B. Baker, Justin L. Shahtout, Piyarat Siripoksup, Quentinn Pearce, Jordan M. Johnson, Fabian Finger, Alexandre Prola, Sarah Pellizzari, Gillian Hale, , Katsuhiko Funai

This study shows that mitochondrial cardiolipin deficiency disrupts electron transport chain function, leading to oxidative stress and promoting MASH in mice and humans.

bioRxiv · Oct 10, 2024Biochemistry