Author

Maria E. Figueroa

University of Miami

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Emmalee R. AdelmaniD· 2 shared Adedamola A. Adele· 1 shared Alan Meeker· 1 shared Anda-Alexandra Calinescu· 1 shared Caio Ricardo Gutierrez Silva· 1 shared Cameron J. Herting· 1 shared Carl Koschmann· 1 shared Charles S. HemenwayiD· 1 shared

Activity by Year

2018-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

Emmalee R. AdelmanORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A ncBAF to cBAF Switch Regulates Human Hematopoietic Progenitor Myeloid Differentiation

Emmalee R. Adelman, Adedamola A. Adele, Caio Ricardo Gutierrez Silva, Katarina Pilala, Liliana Garcia-Martinez, Oriana Teran Pumar, Felipe Beckedorff, Pradeep Reddy Cingaram, Rumela Chakrabarti, Defne Bayık, Ramin Shiekhattar, Lluís Morey, Maria E. Figueroa

This study shows that BRD9 regulates myeloid differentiation in human HSPC by switching ncBAF to cBAF, leading to increased MDSC generation and IL-8 secretion.

bioRxiv · Oct 10, 2026 · PreprintCancer Biology

The chromatin reader protein MLLT1 is critical to maintain normal B lymphopoiesis

Janani Prakash, Nicholas J. Achille, Emmalee R. Adelman, Shubin Zhang, John Hackett Bushweller, Maria E. Figueroa, Charles S. Hemenway, Nancy J. Zeleznik‐Le

This study shows that MLLT1 is essential for normal B lymphopoiesis, as its deletion in mice leads to reduced B cell development and altered gene expression.

bioRxiv · Aug 10, 2026 · PreprintCell Biology

IDH1R132H acts as a tumor suppressor in glioma via epigenetic upregulation of the DNA damage response

Felipe J. Núñez, Flor M. Mendez, Padma Kadiyala, Mahmoud S. Alghamri, Masha G. Savelieff, Carl Koschmann, Anda-Alexandra Calinescu, Neha Kamran, Rohin Patel, Stephen Carney, Marissa Z. Guo, Maria B. Garcia-Fabiani, Santiago Haase, Marta Edwards, Mats Ljungman, , Maria G. Castro

The paper shows IDH1R132H mutation in glioma improves survival and DNA repair by enhancing ATM signaling and radioresistance, which can be reversed by inhibiting ATM or CHK1/2.

bioRxiv · Aug 10, 2018 · PreprintCancer Biology