Author

Mark Bromwich

Yale University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:electronic health records· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Amanda Dabdab BrandelliiD· 1 shared Anson David SingiD· 1 shared Christine A BoweniD· 1 shared Dilpreet KouriD· 1 shared Heike WulffiD· 1 shared Hollis Zeng· 1 shared Imran H. QuraishiiD· 1 shared Jing WuiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperNeuroscience

Amanda Dabdab BrandelliORCID

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2 papers

Recent·Most cited

Kv1.3 inhibition alleviates neuropathology via neuroinflammatory and resilience pathways in a mouse model of Aβ pathology

Rashmi Kumari, Christine A Bowen, Upasna Srivastava, Amanda Dabdab Brandelli, Prateek Kumar, Dilpreet Kour, Sneha Malepati, Wooyoung Eric Jang, Mark Bromwich, Hollis Zeng, Anson David Sing, Steven A. Sloan, Heike Wulff, Srikant Rangaraju

This study shows that Kv1.3 inhibitors reduce neuropathology in a mouse AD model by modulating neuroinflammation, resilience, and neurovascular pathways through distinct cellular mechanisms.

bioRxiv · Dec 25, 2025 · PreprintNeuroscience

Disease-causing Slack potassium channel mutations produce opposite effects on excitability of excitatory and inhibitory neurons

Jing Wu, Imran H. Quraishi, Yalan Zhang, Mark Bromwich, Leonard K. Kaczmarek

The paper investigates how disease-causing gain-of-function mutations in the Slack potassium channel affect excitability differently in excitatory and inhibitory neurons.

Cell Reports · Mar 1, 2024Peer-reviewedNeuroscience