Author

Matthew Drake

King's College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Abigail Saffron TuckeriD· 1 shared Angus J. Lennaárd· 1 shared Annelore WillaertiD· 1 shared Britt HansoniD· 1 shared David J. AdamsiD· 1 shared Francesca Di DomenicoiD· 1 shared Hans J. Friedrichsen· 1 shared Jacqueline K. WhiteiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenetics

Abigail Saffron TuckerORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Toward a model of uORF-mediated translational control: An integrated bioinformatic and experimental approach

Britt Hanson, Nenad Svrzikapa, Ning Feng, Hans J. Friedrichsen, Angus J. Lennaárd, Rushdie Abuhamdah, Nicole Hemmer, Katarzyna Chwalenia, Matthew Drake, Yahya E. Jad, Samir EL Andaloussi, Matthew J. A. Wood, Thomas C. Roberts

The paper combines experimental and computational methods to model how uORFs regulate translation, identifying key features and interactions that influence their repressive activity.

bioRxiv · Jul 28, 2025 · PreprintBiochemistry

Collateral damage: Identification and characterisation of spontaneous mutations causing deafness from a targeted knockout programme

Morag A. Lewis, Neil J. Ingham, Jing Chen, Selina A. Pearson, Francesca Di Domenico, Sohinder Rekhi, Rochelle Allen, Matthew Drake, Annelore Willaert, Victoria Rook, Johanna C. Pass, Thomas Keane, David J. Adams, Abigail Saffron Tucker, Jacqueline K. White, Karen P. Steel

The paper identifies and characterizes spontaneous mutations causing deafness in mice from a targeted knockout program, showing these mutations are a common side effect of intensive breeding.

BMC Biology · Mar 16, 2022Peer-reviewedGenetics