Author

N. Ansari

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Biophysics· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

B. Crimeen-Irwin· 2 shared N. A. Sims· 2 shared T. J. Martin· 2 shared V. Rizzi· 2 shared A. L. Jenner· 1 shared A. P. Browning· 1 shared A. P. Johnston· 1 shared C. Drovandi· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersCell Biology

B. Crimeen-Irwin

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Author profile →

5 papers

Recent·Most cited

OneOPES, a combined enhanced sampling method to rule them all

V. Rizzi, S. Aureli, N. Ansari, F. L. Gervasio

The paper introduces OneOPES, a replica exchange method combining multi-replica simulations and CV-based enhanced sampling to improve phase space sampling in molecular dynamics.

bioRxiv · Mar 6, 2023Biophysics

Identifying cell-to-cell variability in internalisation using flow cytometry

A. P. Browning, N. Ansari, C. Drovandi, A. P. Johnston, M. J. Simpson, A. L. Jenner

The paper develops a mathematical model to quantify cell-to-cell variability in internalisation using flow cytometry data from human B-cell lymphoblastoid cells.

bioRxiv · Nov 24, 2021 · PreprintCell Biology

Water-triggered, irreversible conformational change of SARS-CoV-2 main protease on passing from the solid state to aqueous solution

N. Ansari, V. Rizzi, P. Carloni, M. Parrinello

The paper shows that water molecules trigger an irreversible conformational change in SARS-CoV-2 main protease when transitioning from solid to aqueous state, leading to asymmetric subunit conformations.

bioRxiv · May 21, 2021Biophysics

Dmp1Cre-directed knockdown of PTHrP in murine decidua is associated with increased bone width and a life-long increase in strength specific to male progeny

N. Ansari, T. Isojima, B. Crimeen-Irwin, I. J. Poulton, N. E. McGregor, P. M. Ho, C. S. Kovacs, E. Dimitriadis, J. H. Gooi, T. J. Martin, N. A. Sims

The paper shows that knocking down PTHrP in murine decidua via Dmp1Cre leads to increased bone width and strength in male offspring, suggesting a maternal influence on bone development.

bioRxiv · Jun 9, 2020Cell Biology

Increased autophagy in ephrinB2 deficient osteocytes is associated with hypermineralized, brittle bones

C. Vrahnas, T. A. Dite, N. Ansari, B. Crimeen-Irwin, H. Nguyen, M. R. Forwood, Y. Hu, M. Ikegame, K. R. Bambery, C. Petibois, M. J. Tobin, G. K. Smyth, J. S. Oakhill, T. J. Martin, N. A. Sims

This study shows that ephrinB2 deficiency in osteocytes increases autophagy, leading to accelerated secondary mineralization and brittle bones.

bioRxiv · Mar 1, 2018Cell Biology