Author

N. Gromak

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Cristini· 1 shared B. Stefanovska· 1 shared C. Beghe· 1 shared D. Salamango· 1 shared E. Law· 1 shared E. M. Hammond· 1 shared E. N. Bergstrom· 1 shared F. M. Buffa· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. Cristini

Mutual co-authors in cluster

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2 papers

Recent·Most cited

R-loop homeostasis and cancer mutagenesis promoted by the DNA cytosine deaminase APOBEC3B

J. McCann, A. Cristini, E. Law, S. LEE, M. Tellier, M. Carpenter, C. Beghe, J. Kim, M. C. Jarvis, B. Stefanovska, N. A. Temiz, E. N. Bergstrom, D. Salamango, M. Brown, S. Murphy, , R. Harris

This study shows APOBEC3B regulates R-loop homeostasis and contributes to mutagenesis in cancer by binding R-loops and altering their landscape.

bioRxiv · Aug 30, 2021 · PreprintCancer Biology

Hypoxia-induced SETX links replication stress with the unfolded protein response

S. Ramachandran, T. Ma, N. Ng, I. Foskolou, M.-S. Hwang, P. Victori, W.-C. Cheng, F. M. Buffa, K. Leszczynska, N. Gromak, E. M. Hammond

This study shows hypoxia induces SETX, which protects cells from DNA damage by linking replication stress and the unfolded protein response.

bioRxiv · May 24, 2020Cancer Biology