Author

N. Senoo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

S. M. Claypool· 2 shared A. Barrientos· 1 shared A. Crocker· 1 shared A. Gokhale· 1 shared A. Ivanova· 1 shared A. Levey· 1 shared A. P. Wingo· 1 shared A. R. Lane· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiochemistry

S. M. Claypool

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Conserved cardiolipin-mitochondrial ADP/ATP carrier interactions assume distinct structural and functional roles that are clinically relevant

N. Senoo, D. K. Chinthapalli, M. G. Baile, V. K. Golla, B. Saha, O. B. Ogunbona, J. A. Saba, T. Munteanu, Y. Valdez, K. Whited, D. Chorev, N. Alder, E. R. May, C. V. Robinson, S. M. Claypool

This study investigates the role of cardiolipin interactions in the ADP/ATP carrier using yeast Aac2, showing that disrupting these interactions impairs structure and transport activity, with clinical relevance in ANT1 mutations.

bioRxiv · May 5, 2023Biochemistry

APOE Expression and Secretion are Modulated by Mitochondrial Dysfunction

M. E. Wynne, O. Ogunbona, A. R. Lane, A. Gokhale, S. A. Zlatic, C. Xu, Z. Wen, D. Duong, S. Rayaprolu, A. Ivanova, E. A. Ortlund, E. B. Dammer, N. T. Seyfried, B. R. Roberts, A. Crocker, , V. Faundez

This study shows mitochondrial dysfunction increases APOE expression and secretion in multiple cell types and models, linking mitochondrial health to Alzheimer's disease risk.

bioRxiv · May 11, 2022Cell Biology