Author

O. Bilkei-Gorzo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:proteomics· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Härtlova· 2 shared D. Fabrikova· 2 shared J. Peltier· 2 shared M. Trost· 2 shared A. Hartlova· 1 shared A. K. Daly· 1 shared B. B. A. Raymond· 1 shared B. Topal· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersCell Biology

A. Härtlova

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Lung macrophages utilize cathepsin K-dependent phagosomal machinery to degrade intracellular collagen

A. Härtlova, I. Fabrik, O. Bilkei-Gorzo, D. Fabrikova, M. Öberg, J. Fuchs, C. Sihlbom, M. Göransson

This study identifies cathepsin K as a key determinant of lung macrophage phagosome function, essential for collagen degradation and regulated by TGF-β and cigarette smoke.

bioRxiv · May 16, 2022Cell Biology

The E3 ligase RNF115 regulates phagosome maturation and host response to bacterial infection

O. Bilkei-Gorzo, T. Heunis, J.-L. Marin Rubio, F. R. Cianfanelli, B. B. A. Raymond, J. Inns, D. Fabrikova, J. Peltier, F. Oakley, R. Schmid, A. Härtlova, M. Trost

This study identifies RNF115 as an E3 ligase that regulates phagosomal maturation and host response to bacterial infection through ubiquitylation.

bioRxiv · Jul 13, 2021Biochemistry

Macrophage Scavenger Receptor 1 mediates lipid-induced inflammation in non-alcoholic fatty liver disease

O. Govaere, S. K. Petersen, N. Martinez-Lopez, J. Wouters, M. Van Haele, R. M. Mancina, O. Jamialahmadi, O. Bilkei-Gorzo, P. B. Lassen, R. Darlay, J. Peltier, J. M. Palmer, R. Younes, D. Tiniakos, G. P. Aithal, , A. Hartlova

This study identifies MSR1 as a key mediator of lipid-induced inflammation in NAFLD, showing its role in macrophage activation and potential as a therapeutic target.

bioRxiv · Feb 1, 2020Immunology