Author

P. Finetti

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 5 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 6 Approach:experimental assay· 4 Assay:bulk RNA-seq· 3 Research Focus:cancer & tumor microenvironment· 3 Research Focus:cell signaling & trafficking· 3 Assay:proteomics· 3 Approach:statistical model· 3 Research Focus:gene regulatory network inference· 2

Frequent Co-Authors

F. Bertucci· 6 shared A. Daulat· 2 shared D. Birnbaum· 2 shared J.-P. Borg· 2 shared L. Camoin· 2 shared M. S. Wagner· 2 shared S. Audebert· 2 shared B. G. Debeb· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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6 shared papersCancer Biology

F. Bertucci

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6 papers

Recent·Most cited

The serine/threonine kinase MINK1 directly regulates the function of promigratory proteins

A. Daulat, M. S. Wagner, S. Audebert, M. Kowalczewska, J. Ariey-Bonnet, P. Finetti, F. Bertucci, L. Camoin, J.-P. Borg

This study identifies MINK1 as a kinase that phosphorylates LL5β, a component of the PRICKLE1 complex, promoting cell migration in triple-negative breast cancer and associating with poor metastasis-free survival.

bioRxiv · Sep 2, 2021Cell Biology

Lipocalin 2 promotes inflammatory breast cancer tumorigenesis and skin invasion

E. Schlee Villodre, X. Hu, R. A. Larson, P. Finetti, K. Gomez, W. Balema, S. R. Stecklein, G. Santiago-Sanchez, S. Krishnamurthy, J. Song, X. Su, N. T. Ueno, D. Tripathy, S. Van Laere, F. Bertucci, , B. G. Debeb

This study shows that lipocalin 2 promotes inflammatory breast cancer aggressiveness and skin invasion by enhancing proliferation, migration, and metastasis.

bioRxiv · Mar 18, 2021Cancer Biology

EFA6B regulates a stop signal for collective invasion in breast cancer

R. Fayad, M. Vazquez Rojas, M. Partisani, P. Finetti, S. Dib, V. Virolle, O. Cabaud, M. Lopez, D. Birnbaum, F. Bertucci, M. Franco, F. Luton

This study shows that EFA6B loss promotes collective invasion in breast cancer by reprogramming cell-ECM interactions and activating CDC42, as seen in patient-derived invasive tumors.

bioRxiv · Mar 19, 2020Cancer Biology

Loss of cyclin A2 in murine colonic epithelial cells disrupts colon homeostasis by triggering DNA damage and dysplasia and high cyclin A2 expression is a good-prognosis factor in patients with colorectal cancer

Y. Guo, M. Gabola, R. Lattanzio, C. Paul, V. Pinet, R. Tang, H. Tulari, J. Bremond, C. Maurizy, Q. Da Costa, P. Finetti, F. Boissiere-Michot, C. Lemmers, S. Garnier, F. Bertucci, , M. Hahne

This study shows that loss of cyclin A2 in colonic epithelial cells causes DNA damage, dysplasia, and colon inflammation, while high cyclin A2 expression is linked to better prognosis in colorectal cancer.

bioRxiv · Jul 3, 2019 · PreprintCancer Biology

Genomic landscape of tumor-host interactions with differential prognostic and predictive connotations

J. Roelands, W. Hendrickx, P. J. K. Kuppen, R. Mall, G. Zoppoli, M. Saad, K. Halliwill, G. Curigliano, D. Rinchai, J. Decock, L. Delogu, T. Turan, J. Samayoa, L. Chouchane, E. Wang, , D. Bedognetti

The paper analyzes tumor-host interactions across 31 cancer types using the ICR signature to identify prognostic and predictive implications of immune phenotypes.

bioRxiv · May 8, 2019Cancer Biology

ECT2 associated to PRICKLE1 are poor-prognosis markers in triple-negative breast cancer

A. Daulat, P. Finetti, D. Revinski, M. S. Wagner, L. Camoin, S. Audebert, D. Birnbaum, L. Kodjabachian, J.-P. Borg, F. Bertucci

The paper identifies PRICKLE1 and ECT2 as poor-prognosis markers in triple-negative breast cancer by showing their interaction and role in RAC1 signaling.

bioRxiv · Dec 14, 2018Cancer Biology