Author

P. J. Southern

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:metagenomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. T. Haase· 2 shared J. Anderson· 2 shared L. Schifanella· 2 shared T. Schacker· 2 shared A. Lai· 1 shared A. Schuetz· 1 shared A. Velez· 1 shared B. Howell· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersImmunology

A. T. Haase

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

SARS-CoV-2 infection is associated with intestinal permeability, systemic inflammation, and microbial dysbiosis in hospitalized COVID-19 patients

C. M. Basting, R. Langat, C. A. Broedlow, C. Guerrero, T. D. Bold, M. Bailey, A. Velez, T. Schroeder, J. Short-Miller, R. Cromarty, Z. Mayer, P. J. Southern, T. W. Schacker, S. E. Safo, C. T. Bramante, , N. R. Klatt

This study examines the impact of SARS-CoV-2 infection on intestinal permeability, systemic inflammation, and microbial dysbiosis in hospitalized patients, finding associations between specific microbial changes and disease severity.

bioRxiv · Dec 7, 2023 · PreprintMicrobiology

Productive and latent HIV infections originate in resting CD4+ T cells

S. Wietgrefe, J. Anderson, L. Duan, P. J. Southern, P. Zuck, G. Wu, B. Howell, C. Reilly, E. Kroon, S. Chottanapund, S. Buranapraditkun, C. Sacdalan, N. Tulmethakaan, D. J. Colby, N. Chomchey, , A. T. Haase

This study shows that resting CD4+ T cells are the initial source of both productive and latent HIV infections in the earliest stages of detectable infection.

bioRxiv · Aug 26, 2022 · PreprintImmunology

The Defenders of the Alveolus Succumb in COVID-19 Pneumonia to SARS-CoV-2, Necroptosis, Pyroptosis and Panoptosis

L. Schifanella, J. Anderson, G. Wieking, P. J. Southern, S. Antinori, M. Galli, M. Corbellino, A. Lai, N. Klatt, T. Schacker, A. T. Haase

This paper investigates how SARS-CoV-2 infection leads to the destruction of alveolar type II cells in fatal COVID-19 pneumonia through multiple inflammatory cell death pathways and suggests potential therapeutic strategies.

bioRxiv · Aug 6, 2022Immunology