Author

P. Salomoni

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:statistical model· 2 Approach:benchmark study· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Aziz· 1 shared A. C. Edwards· 1 shared A. E. Grigoriadis· 1 shared A. M. Flanagan· 1 shared A. P. Leite· 1 shared A. Sanz-Moreno· 1 shared B. A. Mock· 1 shared B. L. Pearson· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. Aziz

Mutual co-authors in cluster

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2 papers

Recent·Most cited

PhysiologybioRxivMar 25, 2022

Deep Phenotyping and Lifetime Trajectories Reveal Limited Effects of Longevity Regulators on the Aging Process in C57BL/6J Mice

K. Xie, H. Fuchs, E. Scifo…·Schmidt-Weber

This study uses large-scale phenotyping in mice to show that longevity regulators affect age-sensitive phenotypes before detectable aging begins, rather than slowing aging rates.

DOI 10.1038/s41467-022-34515-y

mouseproteomics

𝕏 PostBluesky

Cancer BiologybioRxivDec 27, 2021

Aberrant paracrine signalling for bone remodelling underlies the mutant histone-driven giant cell tumour of bone.

L. Cottone, L. Ligammari, H. J. Knowles…·A. M. Flanagan

This study identifies aberrant paracrine signaling between osteoprogenitors and osteoclasts driven by H3.3 mutations as a mechanism underlying giant cell tumor of bone and its response to treatment.

DOI 10.1038/s41418-022-01031-x

humanbulk RNA-seq

𝕏 PostBluesky