Author

Qing Tang

Johns Hopkins University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Akshay Salegaonkar· 1 shared Alejandra Macias-GarciaiD· 1 shared Anli Xu· 1 shared Anthony Monti· 1 shared Atli ThorarenseniD· 1 shared Cameron C. TrenoriD· 1 shared Cassie Hei· 1 shared Chi Hua Sarah Lin· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperMolecular Biology

Akshay Salegaonkar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

DCN1 inhibitor induces fetal hemoglobin through self-limited regulation of CUL3 neddylation

Samuel A. Miller, Anthony Monti, Enhua H. Zhou, Chi Hua Sarah Lin, Orna Lynch, Sayani Mukherjee, Cassie Hei, Robert Nicewonger, Colin Diner, Alejandra Macias-Garcia, Kristen A. Marino, Na Li, Ravi K. Ujjinamatada, Akshay Salegaonkar, Sunil Kuppasani, , Sriram Krishnamoorthy

The paper identifies DCN1 as a therapeutic target for beta-hemoglobinopathies by showing its inhibition increases fetal hemoglobin through selective chromatin changes without cytotoxicity.

bioRxiv · Apr 24, 2026 · PreprintMolecular Biology

KAT8 facilitates the proliferation of cancer cells through enhancing E7 function in HPV-associated cervical cancer

Anli Xu, Xiaoming Yang, Junwei Zhao, Shujun Kong, Qing Tang, Xiangzhi Li, Hongmei Qu, Guoyun Wang

This study shows KAT8 enhances E7 function in HPV-associated cervical cancer by promoting its acetylation and interaction with pRb/E2F1, leading to cell proliferation.

Acta Biochimica et Biophysica Sinica · Feb 1, 2025Peer-reviewedCancer Biology

DBT is a metabolic switch for maintenance of proteostasis under proteasomal impairment

Ran‐Der Hwang, Yuning Lu, Qing Tang, Goran Periz, Giho Park, Xiangning Li, Qiwang Xiang, Yang Liu, Tao Zhang, Jiou Wang

The paper identifies DBT as a metabolic regulator that suppresses proteotoxicity by enhancing ubiquitinated protein clearance under proteasomal impairment.

eLife · Sep 10, 2024Peer-reviewedNeuroscience